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Chromosomes that show partial loss or gain in near-diploid tumors coincide with chromosomes that show whole loss or gain in near-triploid tumors: Evidence suggesting the involvement of the same genes in the tumorigenesis of high- and low-risk
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Saito-Ohara F, Imoto I, Inoue J, et al.
PPM1D is a potential target for 17q gain in neuroblastoma.
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原著論文3
Peuchmaur,M, d'Amore, ESG; Joshi, VV, et al.
Revision of the international neuroblastoma pathology classification based on confirmation of favorable and un favorable prognostic subsets of ganglioneuroblastoma, nodular by applying the age-linked morphologic criteria to its neuroblastic
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Okamoto Y, Ozaki T, Miyazaki K, et al.
UbcH10 is the cancer-related E2 ubiquitin conjugating enzyme.
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原著論文5
Ohira M, Morohashi A, Inuzuka H, et al.
Expression profiling and characterization of 4,200 genes cloned from primary neuroblastomas: Identification of 305 genes differentially expressed between favorable and unfavorable subsets.
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Nakamura Y, Ozaki T, Koseki H, et al.
Accumulation of p27KIP1 is associated with BMP2-mediated growth arrest and neuronal differentiation of human neuroblastoma-derived cell lines.
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Miyazaki K, Fujita T, Ozaki T, et al.
NEDL1, a novel ubiquitin-protein isopeptide ligase for Dishevelled-1, targets mutant superoxide dismutase-1.
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Polo-like kinase 1 (Plk1) inhibits p53 function by physical interaction and phosphorylation.
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原著論文9
Allan, EH, Ho PW, Umezawa, A, et al.
Differential potential of a mouse bone marrow stroma cell line.
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原著論文10
Kramer S, Ozaki T, Miyazaki K, et al.
Protein stability and function of p73 are modulated by a physical interaction with RanBPM in mammalian cultured cells.
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原著論文11
Abe M, Ohira M, Kaneda A, et al.
CpG island methylator phenotype is a strong determinant of poor prognosis in neuroblastomas.
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原著論文12
Lin L, Ozaki T, Takada Y, et al.
topors, a p53 and topoisomerase I-binding RING finger protein, is a co-activator of p53 in growth suppression induced by DNA damage.
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原著論文13
Ohira M, Oba S, Nakamura Y, et al.
Expression profiling using a tumor-specific cDNA microarray predicts the prognosis of intermediate-risk neuroblastomas.
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原著論文14
Hanamoto T, Ozaki T, Furuya K, et al.
Identification of protein kinase A catalytic subunit beta as a novel binding partner of p73 and regulation of p73 function.
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原著論文15
Aoyama M, Ozaki T, Inuzuka H, et al.
LMO3 interacts with neuronal transcription factor, HEN2, and acts as an oncogene in neuroblastoma.
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原著論文16
Hosoda M, Ozaki T, Miyazaki K, et al.
UFD2a mediates the proteasomal turnover of p73 without promoting p73 ubiquitination.
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原著論文17
Kaneko Y, Kobayashi H, Watanabe N, et al.
Biology of neuroblastomas that were found by mass screening at 6 months of age in Japan.
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原著論文18
Isono K, Nemoto K, Li Y, et al.
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原著論文19
Machida T, Fujita T, Oo M L, et al.
Increased expression of pro-apoptotic BMCC1, a novel gene with the BNIP2 and Cdc42GAP homology (BCH) domain, is associated with favorable prognosis in human neuroblastomas.
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原著論文20
Niizuma H, Nakamura Y, Ozaki, et al.
Bcl-2 is a key regulator for the retinoic acid-induced apoptotic cell death in neuroblastoma.
Oncogene
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